LESI MEDULA SPINALIS

Majalah Biomorfologi

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Field Value
 
Title LESI MEDULA SPINALIS
 
Creator Maulina, Meutia
Kalanjati, Viskasari Pintoko
 
Subject Spinal cord lesion; traumatic; non traumatic; complete; incomplete; Lesi medula spinalis; trauma; non trauma; komplit; tidak komplit
 
Description Spinal cord  lesion  is one of major causes  of neurological  disability  due to  trauma  and non  trauma. Thisdisability can cause damage and loss of function below the level of lesion and also has adversed effects onseveral body systems. Traumatic spinal cord lesion is the most widely reported incidence compared to nontraumatic spinal cord lesion. The most leading cause of traumatic spinal cord lesion is motor vehicle crash in children, whereas in old age, falls become the main cause. The mechanism of injury influences the type of spinal cord lesion and the degree of neurological deficit. Spinal cord lesions can be classified into completeand incomplete based on the presence or absence of a function that is maintained below the level of lesion.Complete spinal cord lesion shows absence of voluntary movement or sensation below the level of the lesionon both sides, whereas incomplete lesion shows the persistence of the variation of some functions below the level of  lesion. Knowledge on the anatomy of the  spinal  cord  plays an important role  in  the clinicaldiagnosis. The  level of  lesion  at the different  segments of  the spinal cord  determines different  signs  andsymptoms, caused by destruction of segmental tissue and disconnection of ascending and descending tracts above and below the level of lesion. The level of lesion is helpful in predicting deficits of body functions thatmay  occur, in  determining  the next  treatment  and in predicting the  prognosis of the disease. Incompletespinal cord lesion tends to have a better prognosis. Death usually occurs in lesion with multiple trauma. Theleading cause of death is complications due to neurological disability, i.e. pneumonia, pulmonary embolism,septicemia, and renal failure.
 
Publisher Department of Anatomy and Histology, Faculty of Medicine, Universitas Airlangga
 
Contributor
 
Date 2019-11-15
 
Type info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion

 
Format application/pdf
 
Identifier https://e-journal.unair.ac.id/MBIO/article/view/16008
10.20473/mbiom.v26i1.2013.1-5
 
Source Majalah Biomorfologi; Vol 26, No 1 (2013): Majalah Biomorfologi; 1-5
2716-0920
0215-8833
 
Language eng
 
Relation https://e-journal.unair.ac.id/MBIO/article/view/16008/8629
 
Rights Copyright (c) 2019 Majalah Biomorfologi
http://creativecommons.org/licenses/by-nc-sa/4.0
 

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