Effects of cigarette smoking on erythrocyte sedimentation rate, platelet count, total and differential leucocyte counts in adult male smokers: Blood parameters effected by cigarette smoking in males

Annals of Medical Physiology

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Title Effects of cigarette smoking on erythrocyte sedimentation rate, platelet count, total and differential leucocyte counts in adult male smokers: Blood parameters effected by cigarette smoking in males
 
Creator Sultana, Safia
Afsar, Nabila
Jawad, Mohammed
Hazari, Mohammed Abdul Hannan
 
Subject Platelet count
Smoking
Total leucocyte count
Platelet count
Smoking
Total leucocyte count
 
Description Smoking is one of the leading causes of death worldwide. Smokers have higher risk for coronary heart disease, atherosclerosis, acute myocardial infarction, hypertension, clotting disorders, inflammation, respiratory diseases, cancers, etc. A cigarette smoker is exposed to a number of harmful substances. In this study we hypothesized that smoking causes inflammatory reactions and induces hyperthrombic state in the body which may be reflected in erythrocyte sedimentation rate (ESR), total leucocyte count (TLC), differential leucocyte count (DLC) and platelet count values. The purpose of the study was to study the effects of cigarette smoking on erythrocyte sedimentation rate, total leucocyte count and platelet count in adult male smokers and to compare the results with non-smokers and to establish a relationship between the duration and quantity of smoking with the change in ESR, TLC, DLC and platelet count. A cross sectional study was conducted in the department of Pathology on 86 healthy male subjects (smokers=43 and non-smokers=43). ESR was estimated using Westergrens method. TLC, DLC and platelet counts were estimated using HORIBA Pentra ES60 autoanalyser. TLC and basophil counts were significantly higher in smokers than in non-smokers (p<0.05). The mean value of ESR was higher among smokers than non-smokers but it was statistically insignificant. Platelets counts showed no significant difference between smokers and non- smokers. No correlation was observed in various blood parameters and smoking (in pack years). We conclude that smoking initiates an inflammatory response as evidenced from raised TLC, monocyte and basophil counts.
 
Publisher Quench Academy of Medical Education and Research (QAMER) Publishing House
 
Date 2019-03-28
 
Type info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
Peer-Reviewed Article
text
 
Format application/pdf
application/xml
text/html
 
Identifier https://amphysiol.com/index.php/amp/article/view/v3i1.35185
10.23921/amp.2019v3i1.35185
 
Source Annals of Medical Physiology; Vol. 3 No. 1 (2019); 14-18
2456-8422
 
Language eng
 
Relation https://amphysiol.com/index.php/amp/article/view/v3i1.35185/2019_14_18.pdf
https://amphysiol.com/index.php/amp/article/view/v3i1.35185/2019_14_18.xml
https://amphysiol.com/index.php/amp/article/view/v3i1.35185/2019_14_18.html
 
Coverage Worldwide
 
Rights Copyright (c) 2019 Quench Academy of Medical Education and Research (QAMER)
 

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